![]() Signal characteristics of affected areas usually reflect vasogenic edema, with some exceptions: There may be signs of vasospasm or arteritis 3: The affected regions, as outlined above, are hypoattenuating. ![]() The presence of contrast enhancement, no matter the pattern or how avid, does not portend the clinical outcome. Parenchymal infarctions and hemorrhages are associated with posterior reversible encephalopathy syndrome in respectively 10-25% and 15% of cases. Other uncommon patterns of posterior reversible encephalopathy syndrome in <5% include: purely unilateral, or "central" (brainstem or basal ganglia lacking cortical or subcortical white matter involvement), or spinal cord involvement. Both cortical and subcortical locations are affected. Despite its name, however, posterior reversible encephalopathy syndrome can be found in a non-posterior distribution, mainly in watershed areas, including within the frontal, inferior temporal, cerebellar, and brainstem regions 2,19. Typical posterior reversible encephalopathy syndrome manifests as bilateral vasogenic edema within the occipital and parietal regions (70-90% of cases), perhaps relating to the posterior cerebral artery supply.
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. ArchivesCategories |